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dc.contributor.authorFenner, L
dc.contributor.authorAtkinson, A
dc.contributor.authorBoulle, A
dc.contributor.authorFox, M
dc.contributor.authorProzesky, H
dc.contributor.authorZürcher, K
dc.contributor.authorBallif, M
dc.contributor.authorFurrer, H
dc.contributor.authorZwahlen, M
dc.contributor.authorDavies, M
dc.contributor.authorEgger, M
dc.date.accessioned2017-07-19T13:28:46Z
dc.date.available2017-07-19T13:28:46Z
dc.date.issued2017-06-23
dc.date.submitted2017-07-17
dc.identifier.citationHIV Viral Load as an Independent Risk Factor for Tuberculosis in South Africa: Collaborative Analysis of Cohort Studies. 2017, 20 (1):1-7 J Int AIDS Socen
dc.identifier.issn1758-2652
dc.identifier.pmid28691438
dc.identifier.doi10.7448/IAS.20.1.21327
dc.identifier.urihttp://hdl.handle.net/10144/618960
dc.description.abstractChronic immune activation due to ongoing HIV replication may lead to impaired immune responses against opportunistic infections such as tuberculosis (TB). We studied the role of HIV replication as a risk factor for incident TB after starting antiretroviral therapy (ART).
dc.language.isoenen
dc.publisherInternational AIDS Societyen
dc.rightsArchived with thanks to Journal of the International AIDS Societyen
dc.titleHIV Viral Load as an Independent Risk Factor for Tuberculosis in South Africa: Collaborative Analysis of Cohort Studiesen
dc.identifier.journalJournal of the International AIDS Societyen
refterms.dateFOA2019-03-04T13:29:57Z
html.description.abstractChronic immune activation due to ongoing HIV replication may lead to impaired immune responses against opportunistic infections such as tuberculosis (TB). We studied the role of HIV replication as a risk factor for incident TB after starting antiretroviral therapy (ART).


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